Jin-Shun Qi

Name

Jin-Shun Qi

 

Mailing Address

 

 

Dept. of Physiology,

Shanxi Medical University ,

Taiyuan , Shanxi , 030001

E-mail: jinshunqi2006@yahoo.com

Tel: 0351-413-5019 (O); 0351-407-9580 (H)

    

Personal Status  

 

Sex

Male

Date of Birth

February 3, 1955

Place of Birth

Changzhi , Shanxi Province

Marital Status

Married

  

 


Education and Research Experience:

2003.1 - 2006.1

Postdoctoral research in Penn State University , PA , USA ;

Doing researches on  synaptic plasticity in CTZ-induced epileptiform activity

 

2000.9 - 2003.9

Faculty and Ph.D student¡¯s supervisor in Shanxi Medical University ; Engaged in the study of mechanism of Alzheimer¡¯s disease

 

1997.9 - 2000.7

Shanxi Medical University , Ph.D. in Physiology;

Doing researches on the neurotoxic mechanism of amyloid beta- protein in Alzheimer¡¯s disease by membrane patch techniques

 

1987.9 - 1990.7

Tianjin Medical College , Master in Physiology;

Investigating the effects of atrial natriuretic peptide on the electrical activities of paraventricular neucleus and vasopressin  release

 

1977.9-  1981.1

Changzhi Medical College , Bachelor in Medicine

Academic Positions:

2000-present

Professor,Director

Department of Physiology, Shanxi Medical University

 

1992 -1997

Associate Professor

Department of Physiology, Changzhi Medical College

1990 -1992

Lecturer

Department of Physiology, Changzhi Medical College

1981- 1987

Assistant teacher

Department of Physiology, Changzhi Medical College

Memberships in Professional Societies:

2003-present

 

1997-present

member of Scientific Committee of Neuroscience, Chinese Society of physiological Sciences

member of Chinese Society of Physiological Sciences

2000-present

member of Chinese Society for Neuroscience

 

Research Interests

¡¤ Amyloid b-protein and Alzheimer's disease: electrophysiological effects of  amyloid b-protein fragments.

¡¤ Synaptic plasticity in central nervous system: hippocampal long term potentiation in vivo and in vitro.

¡¤ Mechanisms of Epileptogenesis: extrasynaptic receptor regulation.

Recent Publications

1.        Qi J.S., Wang Y., Jiang M., Warren P. and Chen G, Cyclothiazide induces robust epileptiform activity in hippocampal neurons both in vitro and in vivo. J. Physiol. ( London ) 2006 £¬571(3)£º605-618

2.        Qin Y., Qi J.S. and Qiao J.T. Apolipoprotein E4 suppresses delayed-rectifier potassium channels in membrane patches excised from hippocampal neurons. Synapse 2006; 59(2):82-91;

3.        Qi J.S., Ye L. and Qiao J.T., Amyloid beta-protein fragment 31-35 suppresses delayed rectifying potassium channels in membrane patches excised from hippocampal neurons in rats. Synapse 2004; 51(3):165-72

4.        Qin Y., Qi J. S. and Qiao J.T Homocysteine and Alzheimer's disease. Sheng Li Ke Xue Jin Zhan. 2004; 35(1):66-9 (a review)

5.        Nie H., Wang R., Zhang R.X., Qi J.S. and Qiao J.T., Co-expression of protein kinase Cb subspecies in fos protein immunoreactive neurons in rat superficial dorsal horn during formalin test: an immunocytochemical double staining study.  Chin J Neuronsci, 2003, 19(4): 247-251

6.        Hou SW, Qi JS, Zhang Y, and Qiao JT, Spinal antinociceptive effect of agmatine and tentative analysis of involved receptors: study in an electrophysiological model of rats. Brain Res. 2003; 968(2):277-80.

7.        R.X. Zhang, H. Nie, J.S. Qi and J.T.Qiao, Bilateral phosphorylation of transcription factor creb in rat spinal cord induced by unilateral peripheral noxious t\stimuli. Chinese Journal of Neuroanatomy, 2002; 18(1): 1-7

8.        B.F. Han, C. Zhang, J.S. Qi and J.T. Qiao, ATP-sensitive potassium channels and endogenous adenosine are involved in spinal antinociception produced by locus coeruleus stimulation. Acta Physiologica Sinica, 2002; 54(2): 139-144

9.        J.S. Qi, and J.T. Qiao, Amyloid -protein fragment 31-35 forms ion channels in membrane patches excised from rat hippocampal neurons. Neuroscience, 2001£»105(4): 845-852

10.   Qi Jinshun and Qiao Jiantian, Suppression of large conductance Ca2+-activated K+ channel by amyloid -protein fragment 31 -35 in membrane patches excised from hippocampal neurons, Acta Physiologica Sinica, 2001£»53 (3): 198-204

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